Wood preservation facilities, creosote: chapter D, table 4
Table 4. Potential Health Effects of Exposure to Creosote
| Possible health effects | |||
| Exposure Category (Route of Entry) | Type of exposure | Short-term exposure | Long-term exposure |
| Estimated daily intake from various sources (air, water, food - ex. BBQ meat)a | |||
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Varies largely, depending on the different sources of petroleum hydrocarbon or coal-derived products and their uses. The highest concentrations of PAHs in ambient air in Canada were measured at stations located about 1 km from aluminum smelters using the Horizontal Stud Söderberg process in Jonquière and Shawinigan, Quebec. The median concentrations of total PAHs (the sum of 26 compounds) were 693 ng/m3 (mean of 1687 ng/m3) at Jonquière and 435 ng/m3 (mean of 1519 ng/m3) at Shawinigan, with maxima of 10400 and 16 390 ng/m3, respectively (Ringuette et al., 1993). |
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| Eye contact abcd | Direct contact Mist, vapour or splashes |
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| Skin contact abcd | Occasional direct contact with liquid, vapours or mists Significant skin contact with creosote (i.e. total immersion of worker in tank, improper protective measures inside storage tank or retort) |
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Exposure to airborne contaminant or dust ACGIH threshold limit value (TLV) time weighted averages (TWA) ef |
Inhalation of vapours in excess of TLV Benzene-soluble coal tar pitch volatiles: 0.2 mg/m3air (Potential contributions to overall exposure can occur through the cutaneous route, including mucous membranes and eye, either by airborne or, more particularly, by direct contact with the substance.) |
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| Ingestion abcd | Ingestion of creosote through manipulation of contaminated goods (dishes, gum, candy, food, tobacco, liquids) |
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| Chronic symptoms abcd | Repeated exposures |
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